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1. STAT3 mediates ECM stiffness-dependent progression in ovarian cancer NSTL国家科技图书文献中心

Chenchen, Sun |  Xueqian, Qian... -  《Molecular and Cellular Biochemistry》 - 2025,480(1) - 607~620 - 共14页

摘要: crucial regulatory role of STAT3 in the malignant |  the relationship between STAT3 and matrix stiffness |  of STAT3 in vitro under different matrix |  confirmed the differential expression of STAT3/p-STAT3 not |  verified that STAT3 was a mechanically responsive gene
关键词: Biomechanics |  ECM |  Ovarian cancer |  STAT3 |  Stiffness

2. The transcription factor STAT3 and aging: an intermediate medium NSTL国家科技图书文献中心

Shi, Min |  Li, Honyu... -  《Biogerontology》 - 2025,26(2) - 共14页

摘要:, leading to a variety of aging-related diseases. STAT3 is |  cause the activation of STAT3 to varying degrees |  aging process. Drugs targeting STAT3 can treat |  role of STAT3 signaling factors in the pathogenesis |  regulatory role of STAT3 in senescence related diseases is
关键词: Aging mechanism |  STAT3 |  Aging-related diseases

3. Transcription Factor STAT3-Activated LDHB Promotes Tumor Properties of Endometrial Cancer Cells by Inducing MDH2 Expression NSTL国家科技图书文献中心

Shen, Li |  Wang, Juan... -  《Molecular biotechnology》 - 2025,67(2) - 562~574 - 共13页

摘要: transcription 3 (STAT3), LDHB, and MDH2 were predicted through |  property of Ishikawa cells in vivo. STAT3 bound to the |  promoter region of LDHB, and STAT3 silencing-induced |  effects on EC cell phenotypes. STAT3-activated LDHB | The pathogenesis of endometrial cancer (EC
关键词: EC |  STAT3 |  LDHB |  MDH2

4. NLRC5 in Macrophages Promotes Atherosclerosis in Acute Coronary Syndrome by Regulating STAT3 Expression NSTL国家科技图书文献中心

Chen, Jun |  Chen, Guoqin... -  《Cardiovascular toxicology.》 - 2025,25(3) - 365~378 - 共14页

摘要: M1 phenotype. STAT3 was identified as a target of |  STAT3 ubiquitination. Methylation promoted NLRC5 DNA | . STAT3 reduced the effects of sh-NLRC5-mediated M1/M2 |  coronary syndrome by regulating STAT3 expression. This | The mortality rate of cardiovascular and
关键词: NLRC5 |  STAT3 |  Atherosclerosis |  Ubiquitination |  Methylation

5. Isoliquiritigenin attenuates tumor progression and PD-L1 expression by inhibiting the phosphorylation of STAT3 in melanoma NSTL国家科技图书文献中心

Zeng, Haiyan |  Guo, Aoxiang... -  《Medical oncology.》 - 2025,42(4) - Article: 118~Article: 118 - 共10页

摘要: of programmed cell death ligand 1 (PD-L1), STAT3 |  repress the expression of PD-L1 and STAT3 |  with the expression of STAT3. Furthermore, the STAT3 |  expression by repressing the phosphorylation of STAT3 | Isoliquiritigenin (ISL) has been reported with
关键词: Isoliquiritigenin |  STAT3 phosphorylation |  PD-L1 |  Melanoma

6. STAT3-related lncRNAs in colorectal cancer progression; Special focus on immune cell's evasion NSTL国家科技图书文献中心

Saadh, Mohamed J. |  Abd Hamid, Junainah... -  《Pathology Research and Practice》 - 2025,266 - 155810~155810 - 共10页

摘要: of transcription 3 (STAT3) is essential for |  tumor environment, STAT3 is widely overactivated in |  influence important signaling pathways such as STAT3 |  comprehensive overview of the interaction between the STAT3 | Colorectal cancer (CRC) is globally ranked as
关键词: LncRNAs |  STAT3 |  Colorectal cancer |  MiRNAs |  Immune evasion

7. Granulocyte Colony Stimulating Factor Enhances Decidualization Process of Endometrial Stromal Cells Through STAT3/HOXA10 Axis NSTL国家科技图书文献中心

Zhang, Huakun |  Wu, Zhengzhong... -  《Advanced Biology》 - 2025,9(2) - 共9页

摘要: transducer and activator of transcription 3 (STAT3) and the |  STAT3/HOXA10 axis in the action of G-CSF. Results: G |  enhanced STAT3 activation during decidualization |  induction. STAT3 inhibition diminished the effects of G |  that G-CSF increased Hoxa10 expression in a STAT3
关键词: decidualization |  G-CSF |  HOXA10 |  STAT3 |  THESCs |  G‐CSF

8. STING coordinates resolution of inflammation during wound repair by modulating macrophage trafficking through STAT3 NSTL国家科技图书文献中心

Chen, Cheng |  Cai, Xin... -  《Journal of Leukocyte Biology》 - 2025,117(3) - qiae175~qiae175 - 共12页

摘要: capacity through STAT3 activation. Specifically, STING |  STING-STAT3-chemokine signaling combine to delay |  STAT3 to orchestrate inflammatory resolution necessary |  regulates the macrophage trafficking through STAT3 in | Efficient cutaneous wound healing requires a
关键词: inflammation |  macrophages |  STAT3 |  STING |  wound healing

9. Quinazoline-2,4(1H,3H)-dione modulates STAT3 and FOXO3a signaling in HepG2 cells NSTL国家科技图书文献中心

Manoharan, Suryaa |  Murugesan, Krishnasa...... -  《Bioorganic chemistry》 - 2025,157 - 共25页

摘要: as an effective compound against HCC via STAT3 and |  FOXO3a signaling. STAT3 and FOXO3a are two well-known |  interaction with STAT3 and FOXO3a. To validate its | . Gene and protein expression of STAT3 remained at a | Hepatocellular carcinoma (HCC), the most
关键词: Apoptosis |  Chemotherapy |  FOXO3a |  HCC |  Necroptosis |  STAT3 |  Quinazoline

10. Realgar induces apoptosis by inhibiting glycolysis via regulating STAT3 in myelodysplastic syndrome NSTL国家科技图书文献中心

Xue, Tingting |  Tao, Yuchen... -  《Journal of Ethnopharmacology》 - 2025,341 - Article 119339~Article 119339 - 共14页

摘要: involving STAT3, glycolysis, and apoptosis. Hematoxylin | , CETSA, qRT-PCR, WB, apoptosis assays, and STAT3 |  inhibits STAT3 expression. Further investigation using |  directly bind to STAT3. In MDS mice, realgar treatment |  demonstrated that realgar regulates the STAT3 pathway
关键词: Myelodysplastic syndrome |  realgar |  Glycolysis |  STAT3 |  apoptosis
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